Emma Young – New Scientist 14 November 2012
THERE’S a fuzz in my brain and an ache in my gut. My legs are leaden and my eyesight is blurry. But I have only myself to blame. Besides, I have been assured that these symptoms will pass. Between 10 days and three weeks from now, my body will adjust to the new regime, which entails fasting for two days each week. In the meantime, I just need to keep my eyes on the prize. Forget breakfast and second breakfast, ignore the call of multiple afternoon snacks, because the pay offs of doing without could be enormous.
What about Low-Carb Hi-Fat instead?
The above quote come from a New Scientist which article covers the same Eat, Fast and Live Long territory of recent BBC TV programme. It looks as though they have not shed much light in this research due to the same popular pre-conceptions and misconceptions about LDLs.
The usual erroneous use of ‘bad’ cholesterol in association with Low-density lipoprotein hardly is a common error. Although raised blood LDL is associated with heart disease it is not the cause. The cause is sugar damage to the LDL label which prevents it delivering its payload of fats and fat soluble nutrition to the fat-starved organs of the body. The brain needs huge amounts.
The common medical response to our vital organs being LDL starved is to reduce our fat intake. What they should be doing is preventing the sugar-damage to LDL labels allowing our organs to recognise and consume the LDL associated nutrients.
This is what happens when association of LDL with disease is confused with causation. A complete farce which has blinded medical science for 50 years and more!
Eating carbohydrates will generate sugar which insulin will convert into fat.
Eating proteins instead will generate damaging oxides of nitrogen.
Eating fat makes good sense and will not make you fat.
Low-Carb High-Fat diets achieve what fasting achieves without the same hunger!
Liz & Glyn Wainwright 