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Earlier this year I attended a the London WAPF conference in support of Dr Stephanie Seneff and we were fortunate enough to meet Dr Natasha Campbell-McBride. Once Dr Natasha C-M explains what happens to the food you eat and how it affects your health you are empowered to improve your health. She asks you to consider the community that lives within us digesting our food and protecting us from infection and harm.  Once you understand your symbiotic relationship with this microbial community you will respect it, nurture it and take much greater care of what you put in your mouth.

e.g. If food manufacturers treat food to extend its shelf-life (the spoilage bugs can’t survive on it) you have to consider what that does to your internal community of microbial friends when you eat it!

Put You Heart In Your Mouth!

‘Put your Heart in Your Mouth’ – Dr Campbell-McBride

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Why is it news that full dairy products and milk intended to provide the full nutritional requirements of any active growing mammal are really good for you?  Celebrate all meats, full fat dairy products, eggs, and fish as being exactly right nutritionally. I have been reading biochemistry papers since the 1960’s and have yet to find any evidence that these foods have ever caused disease.  On the other hand refined carbohydrates (sugar-damaged proteins aka AGE) are ultimately quite dangerous for our health.  Advanced glycaemic end-products accumulate and cause disease when we spend decades over-indulging our sweet tooth!

Excellent foods!

Cheese ‘could reduce diabetes risk’ – Daily Telegraph

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I think Kate was shocked by what she was reporting – Fat is actually a good thing to eat!  If only people new how much fat and fat soluble nutrients we need to consume from animal sources, and how alien plant oils are to our biochemical systems. If only the medical profession and population at large could speak and understand the basics of biochemistry.

I was hovering over the nuts display in the supermarket, wondering which to buy. I had just interviewed Oliver Selway, a radical diet-and-fitness coach and proponent of a food regimen that does not fear fats. He had told me macadamias were by far the most nutritious nuts to eat, a nut that any dieter will know is forbidden as it is astonishingly calorific. “They’re the fattiest nuts, you know,” a woman next to me said. “They are so bad.” Cheerily, I repeated Selway’s nutritional proposition: that animal and other natural saturated fats from whole foods are good for us. They are what human bodies have known for millions of years…….All natural fats have functions for health: they are not inherently bad…….margarine ‘the devil’s semen’……..told for decades to cut saturated fat from their diet, they replace have replaced it with what some studies suggest is more harmful: refined carbohydrates. 

Fat lot of good by Kate Spicer – Sunday Times UK

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Lynda Collins is a 1960s teenager who dreams of a better life, and of being loved. Not much to ask, is it? Both wealthy Ellen Heywood and snobbish Sheila Stanworth think that Lynda, brought up in a run-down pub in the poorest part of Milfield, isnt good enough to marry their son. Ellen forces her son, Daniel, to give up Lynda, but Dans best friend, John Stanworth defies his Mother and marries the girl Sheila describes as a cheap imitation Marilyn Monroe with no cooking skills.

The Girl who wasn’t Good Enough (eBook) by Liz Wainwright 9781476479903 | WHSmith.co.uk

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MILLIONS who take statins to ward off heart attacks and strokes must cut their dose over fears they are at risk of serious side-effects.

The fact that the raised blood lipids (LDL) associated with CVD is caused by sugar-damaged to the LDL   (Fructose & Glucose) is still a big secret. Cholesterol was never the guilty party but everyone (including many in the medical profession) still hold this myth about cholesterol and completely miss the cause being dietary sugars!

Statins are bad for you!

STATINS IN NEW HEALTH ALERT

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quoted from Zoe Harcombe’s Blog on Traffic Light Food Labelling

All real foods in the table above have at least one red/amber traffic light warning and olives and cheese have three. The former being a good source of natural fat – especially the much eulogised mono-unsaturated fat – and the latter being an excellent source of calcium and the other bone nutrients vitamin D and phosphorus.

With one exception, all processed foods in the table above have green lights for fat, saturated fat and sugar. Multi grain bread gets an amber for fat content – because of the highly nutritious seeds that is contains. White bread scores better than multi grain.

Zoe

The law of unintended consequences

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Statins reduce our ability to make vital cholesterol. Cholesterol is used to make memory connections in the brain.  The linked paper is worrying for all statin users.

Synapses

Synaptogenesis and neural cholesterol
Nowhere is the impact of cholesterol depletion more keenly studied than in the neurologic arena.  The work of Pfrieger et al. described the functional role of cholesterol in memory through synapto-genesis [24]. Mauch et al. [25] reported evidence that cholesterol is vital to the formation and correct operation of neurons to such an extent that neurons require additional sources of cholesterol to be secreted by glial cells. A recent mini-review by Jang et al. describes the synaptic vesicle secretion in neurons and its dependence upon cholesterol-rich membrane areas of the synaptic membrane [26]. Furthermore, working on rat brain synaptosomes, Waseem [23] demonstrated that a mere 9.3% decrease in the cholesterol level of the synaptosomal plasma membrane could inhibit exocytosis. These data might be particularly worrisome for lovastatin and simvastatin which are known to cross the blood brain barrier [27].
In fact, the proposed use of statins as a thera-peutic agent in Alzheimer’s disease (AD) [28] counters Pfrieger’s evidence [24]. Indeed, a reduc-tion in cholesterol synthesis leads to depletion of cholesterol in the lipid rafts – i.e. the de-novo cholesterol is required in the neurons for synaptic function and also in the neuronal membrane fusion pores [29].
Cognitive problems are the second most frequent type of adverse events, after muscle complaints, to be reported with statin therapy [30] and this has speculatively been attributed to mitochondrial effects. The central nervous sytem (CNS) cholesterol is synthesised in situ and CNS neurons only produce enough cholesterol to survive. The substantial amounts needed for synaptogenesis have to be supplemented by the glia cells. Having previously shown that in rat retinal ganglion cells without glia cells fewer and less efficient synapses could form, Göritz et al. [31] indicate that limiting cholesterol availability from glia directly affects the ability of CNS neurons to create synapses. They note that synthesis, uptake and transport of cholesterol directly impacts the development and plasticity of the synaptic circuitry. We note their very strong implication that local de-novo cholesterol synthesis in situ is essential in the creation and maintenance of memory.
There should be further consideration of cholesterol depletion on synaptogenesis, behaviours and memory loss for patients undergoing long-term statin therapy. This is particularly important with lipophilic statins which easily cross the blood brain barrier [32].
The effects of statins on cognitive function and the therapeutic potential of statins in Alzheimer’s disease are not clearly understood [28]. Two randomised trials of statins versus placebo in relatively younger healthier samples (lovastatin in one, simvastatin in other) showed significant worsening of cognitive indices relative to placebo [33, 34]. On the other hand, two trials in Alzheimer samples (with atorvastatin and simvastatin respectively) suggested possible trends to cognitive benefit, although these appeared to dissipate at 1 year [35, 36]. A recent Cochrane review concluded that there is good evidence from randomised trials that statins given in late life to individuals at risk of vascular disease have no effect in preventing Alzheimer´s disease or dementia [37]. However, case reports and case series from clinical practice in the real world reported cognitive loss on statins that resolved with discontinuation and recurred with rechallenge [30].

Statin Use Increases Dementia Risk

The ‘statins cure everything’ idea exposed

Statins are said to be useful against more than heart disease, e.g. cancer, lung disease, heart failure, hip fractures and much more. The way in which researchers have studied these alleged benefits is confounded with a serious error. As an example I shall analyse the allegation that statin treatment prevents Alzheimer’s disease.  The idea goes against common sense. Today we know that not only is the brain the cholesterol-richest organ in the body; cholesterol is also vital for its function, because the creation of nerve impulses demands a steady production of cholesterol.

Uffe Ravnskov

Book Cover

Statin treatment accelerates atherosclerosis – Uffe Ravnskov

Recently two separate studies showed that people on statin treatment develop atherosclerosis more often than untreated people. One of them was published in Diabetes Care, the other one in Atherosclerosis. You can get more details about this shocking finding in Dr. Mercola´s interview with senior scientist researcher Stephanie Seneff from MIT, who also is a member of THINCS. Read also Dr. Mercola´s comments about statin treatment.

Uffe Ravnskov, MD, PhD,

Independent Investigator
Spokesman of THINCS

The International Network of Cholesterol Skeptics

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If only we could have all the data on ‘Statins’, I have good reasons to suspect that they will become the pharmaceutical industry’s ‘PPI & LIBOR scandal’. In his latest book ‘Bad Pharma’ Ben Goldacre exposes the ways in which the trials of drugs can be used to give us the headline good news whilst toxicity, adverse events and important data about ‘all cause mortality’ is well hidden. Even (especially) the Doctors aren’t told. This book is on my reading list now.  This links to the Guardian review by Luisa Dilner.

Bad Pharma by Ben Goldacre – book review