The authors’ findings suggest that higher levels of cholesterol are associated with a better outcome in the early phase after ischemic stroke.
Department of Neurology, Centre Hospitalier Universitaire Vaudois, Lausanne, Switzerland.
More bad news for piles up for statins.
Compared to the individuals not taking statins, those taking statins had higher prevalence of risk factors and obstructive CAD……
Compared to individuals not on statin therapy, individuals who were taking statins were older and had higher body mass index (BMI), risk factors, lower LDL ….& lower HDL….
Statin use was associated with a higher frequency of severe coronary artery stenoses as well as numbers of coronary vessels with obstructive coronary artery disease. Further, statin use was associated with a differentially increased prevalence and extent of mixed-plaque and calcified plaque but not non-calcified plaque.
….the use of statins was associatedwith……..increasing presence and numbers of coronary segments with calcified plaque components.
Many references to contradictions in previous studies and use the word ‘surrogate’ seem to suggest that those who determine medical protocols are going to have problems coming to terms with these disturbing findings.
Statin use is associated with an increased prevalence and extent of coronary plaques possessing calcium.
There has been recent speculation about the benefits of blood letting in Type 2 diabetes. However in 2008 L. Mascitelli, F. Pezzetta, M. R. Goldstein wrote
…dietary factors, which inhibit iron absorption (i.e. low-fat dairy consumption and habitual coffee intake), have been shown to be associated with a substantially lower risk of type 2 diabetes
They went on to conclude that a few basic lifestyle interventions might make more sense……
…we suggest that, in patients with or at high risk for type 2 diabetes, improving insulin sensitivity with physical exercise, metformin therapy and low-carbohydrate diet might break the vicious circle of insulin resistance, increased intestinal iron absorption and further worsening of insulin resistance.
...the enduring quest for cholesterol lowering in humans in order to reduce cardiovascular risk may have unsuspected side effects. They may not be relevant for the majority of patients, and are therefore not reported in large statin trials, but may lead to adverse effects in patients with an underlying susceptibility to hematological disorders.
Our own review concludes a minimum of 20% molecular cholesterol is functionally important in all cell types.
Alzheimer’s disease is a devastating disease whose recent increase in incidence rates has broad implications for rising health care costs. Huge amounts of research money are currently being invested in seeking the underlying cause, with corresponding progress in understanding the disease progression. In this paper, we highlight how an excess of dietary carbohydrates, particularly fructose, alongside a relative deficiency in dietary fats and cholesterol, may lead to the development of Alzheimer’s disease. A first step in the pathophysiology of the disease is represented by advanced glycation end-products in crucial plasma proteins concerned with fat, cholesterol, and oxygen transport. This leads to cholesterol deficiency in neurons, which significantly impairs their ability to function. Over time, a cascade response leads to impaired glutamate
signaling, increased oxidative damage, mitochondrial and lysosomal dysfunction, increased risk to microbial infection, and, ultimately, apoptosis. Other neurodegenerative diseases share many properties with Alzheimer’s disease, and may also be due in large part to this same underlying cause.
Dr Stephanie Seneff is a senior research scientist at MIT.
Alzheimer’s disease: The detrimental role of a high carbohydrate diet
Every individual gets at most only one chance to grow old. When you experience your body falling apart, it is easy to imagine that this is just due to the fact that you are advancing in age. I think the best way to characterize statin therapy is that it makes you grow older faster. Mobility is a great miracle that cholesterol has enabled in all animals. By suppressing cholesterol synthesis, statin drugs can destroy that mobility. No study has shown that statins improve all-cause mortality statistics. But there can be no doubt that statins will make your remaining days on earth a lot less pleasant than they would otherwise be.
To optimize the quality of your life, increase your life expectancy, and avoid heart disease, my advice is simple: spend significant time outdoors; eat healthy, cholesterol-enriched, animal-based foods like eggs, liver, and oysters; eat fermented foods like yogurt and sour cream; eat foods rich in sulfur like onions and garlic. And finally, say “no, thank-you” to your doctor when he recommends statin therapy.
Dr Stephanie Seneff is a senior research scientist at MIT.
How Statins Really Work Explains Why They Don’t Really Work.
I really enjoyed this book. It was different from my usual read as I normally go for thrillers. The book follows the title character `Lynda Collins’ from early adolescence into her 30s. It is full of strong and colourful characters and I found it a bit like reading a soup opera. If you like Eastenders or Coronation Street then you will enjoy this book. I look forward to book 2.
Mrs. Joyce M. Scott
Reviews: The Girl who wasn’t Good Enough (The Lynda Collins Trilogy)
The diet was hatched in Poland some 40 years ago by Dr. Jan Kwasniewski, who started developing it while working as a dietician for a military sanitarium in Ciechocinek, Poland. There he observed that many of his patients were sick, “not because of any pathogenic factors … but the result of one underlying cause – bad nutrition,” according to his English language “Optimal Nutrition” book. After experimenting on his family and himself, Kwasniewski concluded that the ideal nutritional combo came from eating three grams of fat for every one gram of protein and half a gram of carbohydrates.
Petro Dobromylskyj
Petro Dobromylskyj
Liz & Glyn Wainwright 